In autologous fat grafting, graft survival is decided less by how the fat is injected than by the tissue it is injected into. Rigottomia (rigottomy) is the percutaneous manoeuvre that reconditions that recipient site — releasing fibrosis, expanding volume and increasing vascular access — so the seeded graft has somewhere to live. This is a professional overview of how rigottomia prepares the recipient bed and why it complements clean fat grafting technique.

The recipient site is the rate-limiting factor

Grafted adipocytes survive by diffusion until neovascularisation reaches them, typically within the first days. Anything that increases the distance between a fat parcel and a perfused surface — large boluses, high injection pressure, or a stiff, hypovascular bed — pushes graft centres beyond the reach of diffusion and drives resorption and, in the worst case, oil cysts and fat necrosis. In fibrotic, irradiated or scarred tissue the bed itself is the constraint: it is non-compliant, offers little interstitial space, and is poorly vascularised.

What rigottomia does mechanically

Passing a fine needle or blade percutaneously, the surgeon performs multiple small releases (classically around 2 mm) through the restrictive fibrous septa and subdermal aponeurosis. The effect is threefold:

The goal is a vascularised, compliant scaffold of micro-cavities to be seeded with micrografts — not an empty pocket to be filled.

Rigottomia within a recipient-site conditioning strategy

Percutaneous release is most powerful as one element of a wider conditioning approach, popularised by Rigotti and Khouri. External tissue expansion applied before grafting increases recipient-site vascularity and interstitial volume; percutaneous rigottomia releases the aponeurotic restraint that limits that expansion; and structural, low-pressure micrografting distributes the graft as fine parcels through the prepared bed. Percutaneous aponeurotomy plus lipofilling has been described in the literature as a regenerative alternative to flap reconstruction in selected defects for precisely this reason.

Graft quality still matters

A well-prepared bed cannot rescue a poorly processed graft. Gentle, low-negative-pressure harvest and processing that removes blood, oil and cellular debris while preserving adipocyte integrity and the regenerative stromal vascular fraction give the seeded parcels their best chance. Closed-system washing and concentration — the principle behind fat dialysis and systems such as EXAFAT — is aimed at exactly this: cleaner, better-preserved fat to seed into the released matrix. Recipient-site preparation and graft quality are complementary levers, not alternatives.

Patient selection and judgement

Rigottomia is most valuable where the recipient bed is the problem: mature scars, burn contractures, irradiated skin, tethered reconstruction sites and congenital fibrous constriction. It is technique-dependent — too aggressive and confluent a release risks macrocavities and vascular compromise; too timid and the fibrosis is not meaningfully released. As with all grafting, staged sessions are often more predictable than attempting excessive volume in a single pass, and outcomes depend on tissue quality, technique and patient factors.

The bottom line

Rigottomia reconditions the recipient site — releasing fibrosis, expanding volume and improving vascular access — so that structural fat grafting can succeed in tissue that would otherwise reject it. Paired with clean, well-preserved fat, it is a core tool for grafting into scarred, irradiated and constricted beds. For the processing side, see our overview of fat processing quality and fat dialysis.

This is a general professional overview of a surgical technique, not clinical guidance or a treatment protocol. Follow current evidence, device instructions for use and local governance.